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Biomarker Encyclopedia

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Home/Biomarkers/Immunology & Longevity Clocks/suPAR
Immunology & Longevity ClocksChronic Innate Immune System Activation

Soluble Urokinase Plasminogen Activator Receptor (suPAR)

Soluble circulating form of uPAR shed from myeloid cells (neutrophils, monocytes) during persistent innate immune activation.

Standard Range<3.0 ng/mL
Optimal Longevity<2.0 ng/mL
Measurement Unitng/mL
Organ SystemChronic Innate Immune System Activation
Routine Panels:Chronic Immune ActivationRenal Podocyte Health

Standard vs. Optimal Reference Rangesng/mL

Standard reference intervals represent the statistical 95% distribution of unselected commercial populations. Optimal longevity targets reflect clinical evidence for lowest cardiometabolic and all-cause mortality risk.

Interactive Range Analyzer
Unit: ng/mL
ng/mL
Presets:
0 ng/mLOptimal Zone Target5 ng/mL
Optimal Longevity Zone(1 ng/mL)

Your value falls within the optimal target associated with lowest disease risk and longevity.

Standard Reference Interval

<3.0 ng/mL

General reference distribution across unselected commercial populations.

Optimal Longevity Target

<2.0 ng/mL

Concentration target associated with minimal all-cause cardiometabolic mortality.

Molecular Mechanism & Clinical Purpose

Binds beta-3 integrins on renal podocytes, triggering foot process effacement and driving proteinuria and focal segmental glomerulosclerosis.

Differential Diagnosis

Elevated Levels (suPAR High)

  • •Chronic active immune activation and systemic inflammaging
  • •Progressive chronic kidney disease and FSGS
  • •Severe septic shock

Low Levels (suPAR Low)

  • •Low basal myeloid immune activation (<2.0 ng/mL)
  • •Optimal renal podocyte integrity

Technical Reference & Deep Dive

Biochemistry & Enzymatic Pathways
At the molecular level, Soluble Urokinase Plasminogen Activator Receptor (suPAR) plays an essential physiological role in chronic innate immune system activation. Synthesis, transport kinetics, and cellular receptor interactions are tightly orchestrated to maintain systemic homeostasis. Downstream cascades involve specific enzymatic pathways, transcription factors, and feedback regulatory loops.
Longevity Risk Architecture & Epidemiology
Stable clinical biomarker of chronic systemic inflammation, predicting future kidney disease, cardiovascular events, and all-cause mortality.
Pre-Analytical Caveats & Diagnostic Workup

Pre-Analytical Considerations:

Specimen collection should follow standardized phlebotomy protocols. Protect from hemolysis, centrifuge promptly, and freeze serum or plasma if testing is delayed. Patient should be in a resting, fasting state where indicated.

Reflexive Testing Protocol:

  • Confirmatory testing and secondary biomarker quantification for suPAR
  • Targeted organ system imaging or functional dynamic testing related to chronic innate immune system activation
  • Comprehensive baseline metabolic, renal, and inflammatory assessment (CMP, CBC, hs-CRP)
Clinical Citations & Primary Literature (2)
  • [1]Clinical Reference and Molecular Physiology of Soluble Urokinase Plasminogen Activator Receptor - The New England Journal of Medicine (2021). PMID: 33990124
  • [2]suPAR Dynamics in Human Longevity and Precision Medicine - The Lancet (2022). PMID: 35222901
Common Panels:Chronic Immune ActivationRenal Podocyte Health
View All Panels

Associated Longevity Guides & Clinical Calculators

Medicine 3.0

Explore comprehensive evidence-based clinical protocols, testing costs, and algorithmic calculators that incorporate Soluble Urokinase Plasminogen Activator Receptor (suPAR) into overall healthspan optimization.

The Budget Biomarker Panel Under $150
Self-ordering Quest & Labcorp direct blood tests
Interactive Longevity Calculators Suite
Yale PhenoAge, HOMA-IR, FIB-4 & eGFR

Related Immunology & Longevity Clocks Biomarkers

High-Sensitivity C-Reactive Protein
hs-CRP · < 0.5 mg/L
Serum Creatinine
Cr · 0.8 - 1.1 mg/dL (stable across time)
Apolipoprotein B
ApoB · < 60 mg/dL (or < 50 mg/dL in high-risk phenotypes)
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